Up to 43% of the population struggles with addiction-like eating — nearly half the country. And for decades, the entire industry has had the definition wrong.
By Leslie Chen | Founder of Rise Lean | MSc Applied NeuroscienceThe Cafeteria
I find myself sitting in the college cafeteria and this is my 3rd bagel. I didn’t know how to stop myself from wanting it. After the first, I felt full.
As I was putting that bagel into my mouth, I was grieving the fact that just three months ago, before I came to America and got into this whirlwind of a food nightmare, I couldn’t even finish 2/3 of the same bagel.
Now I feel I’m losing myself. Not just my healthy relationship with food which I’ve had for the past 19 years, but also my body — in the past 3 months, I’ve put on 30 pounds, my sanity — as I negotiate with myself around food all my waking hours, and my study — if I couldn’t maintain my GPA, I’d lose my scholarship. I’m at the edge of it.
That was me 20 years ago. I didn’t know the Yale Food Addiction Scale existed. I didn’t even know food addiction was a thing. I put everything under the same umbrella — a discipline and self-control problem that’s causing weight gain and toxic eating behaviors. When I googled for a solution, I didn’t search “food addiction” but “weight loss” — as if this eating nightmare would disappear after my weight became normal.
That was the wrong problem. And the industry is still making the same mistake.
Now, 20 years later, I’m the leading expert in the food addiction recovery space, with a neuroscience background and a clear understanding of the key brain mechanisms that make this happen.
I’ve spent years reading, analyzing, and writing papers on it.
I’ve drawn diagrams mapping the major neural systems underlying constant, impulsive, addictive eating. Before this, there wasn’t a comprehensive, integrated image that put everything together and depicted the full brain mechanism at work that hosts this problem — even in the neuroscience world, despite hundreds of published studies addressing different angles and neurobiological elements of the problem.
And in the past 10 years, I’ve helped hundreds of people cleanly solve this problem. My clients range from the general population to general managers, leading scientists, physicians, unicorn entrepreneurs, therapists, and world-ranking athletes who are living textbooks of discipline but still couldn’t hold independence and sovereignty in front of food.
What compels me to write this article is this: the world has food addiction completely wrong. Wrong definition. Wrong treatment. For decades. And people are paying for that ignorance with their entire lives.
My oldest client right now is in her 70s — more than half a century of struggle.
Unfortunately, for most people, the struggle doesn’t go away. It only gets reinforced. Ignorance on this subject matter costs a lifetime of peace.
Food addiction has a clear scientific framework. It has for over fifteen years. The Yale Food Addiction Scale — introduced in 2009 and updated in 2016 — measures it using criteria directly adapted from substance use disorder in the DSM-5: loss of control, continued use despite harm, failed attempts to cut back, tolerance, withdrawal. It is not yet a formal DSM-5 clinical diagnosis. That gap is not a sign the science is weak. It is a sign the institution hasn’t caught up.
Here is the definition the industry keeps getting wrong:
Food addiction is not simply an eating disorder. It is not simply an addiction disorder.It is both — simultaneously.
It carries the behavioral patterns of an eating disorder and the neurological patterns of a substance addiction disorder.
That combination puts it in its own category entirely. And that category has been almost completely ignored by the solution landscape.
This is not a technicality. It is the entire reason why most people who struggle with food addiction never get better — not because they haven’t tried hard enough, but because they have solved the wrong problem the entire time.
By strict YFAS criteria, food addiction affects 16–20% of the US population (Penzenstadler et al., 2019). When you include people who self-report addiction-like patterns without meeting the full diagnostic threshold, that number rises to 43% (Leary et al., 2021).
That is not a niche problem. That is almost half the country.
And yet the dominant conversation around food addiction calls it a discipline problem. A mindset problem. A food choice problem. Occasionally — in the more sophisticated corners of the wellness space — a “reward loop” problem, though rarely with enough depth to mean anything actionable.
Here is what it actually is.
Here’s what the industry doesn’t understand:
Food addiction is not one thing going wrong. It is several neural systems, simultaneously compromised, co-creating a self-reinforcing loop that behavioral willpower is simply not designed to break.
Let me walk you through the architecture, without the jargon.
The mesolimbic dopaminergic pathway — the brain’s primary reward circuit — becomes over-activated in response to certain foods.
This pathway doesn’t just generate pleasure. It generates motivation. When it’s dysregulated, food stops being something you enjoy and becomes something your brain treats as a high-priority target, worth pursuing, worth effort, worth the guilt afterward.
This is the source of “I crave food too much!”The orbitofrontal cortex — the top-down regulatory mechanism that evaluates decisions and applies the brakes — shows impaired function in individuals with food addiction (Kalon et al., 2016; Gearhardt et al., 2011).
When the reward accelerator is pressed hard and the control brake is compromised, the outcome is not a character flaw. It’s a predictable neurological result.
This is the source of “I can’t control myself.”With repetition, the nucleus accumbens accumulates ΔFosB — a protein that functions as a molecular memory marker (Nestler, 2008). Its message to the brain is something like: When this person encounters X, make her eat.
The behavior stops being a choice and starts being an automatic execution of a neural instruction like a software program. This is what “compulsive” actually means at the biological level. It is not weakness. It is a hardwired program running in the background.
This is the source of “I give into food every single time.”The amygdala and hippocampus — emotion and memory centers — become tightly interlaced in individuals with food addiction (Kalon et al., 2016). Specific memories become charged with emotional weight, creating an associative network that the brain draws on constantly. Stress at work links to the bowl of chips. Loneliness links to the ice cream at midnight. Celebration links to the binge. The constant overthinking, too. These are not habits in the ordinary sense. They are neurologically reinforced emotional predictions.
Interoception — your ability to accurately read your own internal state, including hunger and fullness — is significantly impaired in people with disordered eating. Research across 41 studies and nearly 8,000 participants found the effect size of this impairment to be 1.62 (Jenkinson et al., 2018) — an enormous, remarkably consistent gap.
You are not imagining it when you genuinely can’t tell whether you’re hungry. Sometimes, you may even hallucinate hunger. Your brain’s internal signal processing is disrupted, and food addiction sits squarely inside that disruption.
This is the source of “I don’t even know what fullness really is anymore.”Without interoception working properly, overeating often becomes the default everyday experience with or without addiction.
And here is the piece that almost no one is talking about — and it’s the biggest piece holding everything:
The brain is, at its core, a prediction machine. It doesn’t experience reality neutrally — it constantly generates forward projections and then works to minimize the gap between prediction and actual experience.
When your brain has built a robust model of you as “someone who always gives in to chocolate” or “someone who loses control around food,” it doesn’t just describe that identity. It enforces it — through predictive coding, through what it attends to, through how it interprets internal signals, through what behaviors it primes you toward (Critchley and Garfinkel, 2017; Seth, 2013).
This is the source of the ultimate self-definition: “I have no power in front of food.”And that single belief holds the full-blown physical and emotional experience of someone who is guilty, anxious, disappointed, out of control around food, with a compromised relationship with their own self — and it becomes the ground they navigate the rest of their life from.
So no — food addiction is not one thing going wrong. It’s a web of neurons wiring and firing together among several neural networks sustaining pre-programmed experiences.
It’s one problem covering eating disorder and addictive disorder.
That’s the true definition of food addiction. It isn’t in your head. It’s in your brain.
Discipline, diets, mindset problems are too tiny to define it.
When you can’t define the problem, you can’t solve the problem.
In fact, the dominant, widespread solutions of the problem are far from matching the definition of it, which I’m going to talk about next.
Let me say the most uncomfortable thing first: for a very long time, there was almost no solution on the market that was actually built for food addiction. That gap is what drove me to build Lean Instinct Formula™ in the first place.
Even today, dedicated, viable solutions to food addiction are still very rare — not because the problem is new. Because the industry, without fully understanding the problem, never properly named it.
Remember — food addiction is both an eating disorder and a substance addiction disorder, at the same time. So when you go looking for help, here’s what you actually find: solutions built for one half of the problem, or the other, or neither. Almost nothing built for the thing itself.
Look at how few solutions even exist. Leary et al. (2021) did a systematic review of food addiction interventions, a search that surfaced 16,649 peer-reviewed studies from 2008 to 2020 using keywords including food addiction, eating behaviors, interventions, measuring food addiction, etc. After combing the entire field, they could only find nine studies that even measured food addiction as an outcome — and in every single one of the nine, food addiction wasn’t even the point. It was a secondary measurement, tacked onto studies designed for something else: weight loss, bariatric surgery, smoking cessation. Not one single solution across the nine studies was actually built to treat food addiction.
16,649 peer-reviewed studies. Nine relevant interventions. Zero dedicated solutions.
16,649 → 9 → 0.
Zero. For decades, not one single solution was developed to solve food addiction.
(By 2020, the Lean Instinct Formula™ hadn’t yet been established long enough to be studied.)
And the handful of things people do get handed? Every one of them was designed for a different problem.
Therapy (CBT-E) is the gold standard — for eating disorders. Not food addiction. Yes, the two overlap. But food addiction isn’t just an eating disorder, so reaching for CBT-E here is grabbing a tool built for a different job. And even on its home turf, it’s not impressive: less than 50% remission in binge-eating (Moberg et al., 2021), with 30% or more relapsing within a year (Bergh et al., 2017). It reshapes some thinking patterns. It doesn’t restore your ability to feel full. It doesn’t touch cravings and impulsivity at the neural level. It doesn’t restore the brain’s top-down control system. It doesn’t go anywhere near the prediction engine quietly running the whole thing.
Ozempic and the other GLP-1 drugs were built for weight and metabolic conditions — not food addiction. The quieting of cravings is a side effect, not the target. And yes, that side effect is real: they promote fullness and dampen the reward pathway (Tongta et al., 2025). But they don’t touch the learned patterns, the emotional wiring, the prediction engine, or your sense of self. The moment the drug stops, it all comes back. That’s not treatment. That’s a pause on a problem that’s still running underneath.
Restriction programs — this is the one that shows how seriously ignorant the industry is about food addiction.
The reality is: food restriction makes food bigger and addiction stronger.
Here’s the mechanism, and it’s the exact opposite of what these programs assume — not the flat, plain you-want-what-you-can’t-have explanation:
Fear and anxiety are core components of addiction. And when the brain registers fear and anxiety around something, it does what brains are built to do — it tries to avoid that thing. So you restrict. You white-knuckle. You build your whole day around not eating the food. But every time you execute that avoidance — the natural downstream outcome of the upstream emotion, you’re confirming the upstream emotion — the very fear and anxiety that drive the addiction in the first place.
The downstream behavior reinforces the upstream emotion. That’s how addiction gets stronger.
In the world of psychiatry and neuroscience, this isn’t a secret but is openly discussed and proven in many studies. I have lost count of the clients who came to me after years in restriction-based programs that claimed to treat food addiction. All those programs did was deepen the obsession and leave the underlying wiring completely intact, if not tighter.
Now, back to the solution umbrella as a whole, the pattern is the same across all of them: wrong category, wrong mechanism, symptoms managed on the surface while the engine keeps running.
Trying harder feels exactly like pushing a car with the engine off.
Most people with food addiction and food noises are simply told to try harder. But without touching what’s actually driving the whole web — the neural patterns, the emotional wiring, the interoception, the identity predictions — trying harder feels exactly like pushing a car with the engine off. You can throw your whole body into it. The car isn’t going to move on its own.
We have 20–43% of the population struggling with food addiction or addictive eating patterns. We have science that has clearly mapped the neurobiology of this problem for over fifteen years. And we have an industry that is still selling discipline reframes, meal plans, and fear-based restriction while calling it a solution.
The people who come to me believing they are broken — that they lack willpower, that something is fundamentally wrong with them — are not broken. They have a real neurological problem that real science has described in precise detail. They have been handed the wrong map and told to walk harder.
I’ve worked with many clients in their sixties who have been stuck in the same cycle and misled by the wrong treatments for 50 years, sacrificing joy, peace, self-image, and often time, opportunities and potential.
When I speak to a client who thinks she’s broken, grieving every day she has lost to food, what I’ve shared here today is the first thing I share with her.
Evidence Base
The claims in this piece are grounded in peer-reviewed research. Full citations below, in the order they support the argument above.